Muestra la distribución de la producción WoS, Scopus y SciELO del autor.
Publicaciones WoS (Ediciones: ISSHP, ISTP, AHCI, SSCI, SCI), Scopus, SciELO Chile.
| Firmas del autor | |
| Nombre | CANALES-MARTINEZ, CESAR PATRICIO |
| Género | Hombre |
| Área Principal WOS | Biochemistry & Molecular Biology; Biology; Multidisciplinary Sciences; |
| Afiliación Principal | Centro De Estudios Científicos |
Publicaciones en Chile
Citas Totales
Afiliaciones Chilenas
| WOS | #Pub |
|---|---|
| Biochemistry & Molecular Biology | 2 |
| Biology | 1 |
| Multidisciplinary Sciences | 1 |
| Genetics & Heredity | 1 |
| Scopus | #Pub |
|---|
| SciELO | #Pub |
|---|
| Autor | Género | # Pub |
|---|---|---|
| MOLINA-ARAVENA, JESSICA ALEJANDRA | Mujer | 3 |
| CARMONA-MORA, PAULINA | Mujer | 3 |
| YOUNG, JUAN I. | Hombre | 2 |
| CARPIO, J. DANIEL | - | 1 |
| KAIRATH-OLIVA, PAMELA ANDREA | Mujer | 2 |
| ENCINA-OLAVARRIA, CAROLINA ANDREA | Mujer | 1 |
| WALZ, KATHERINA | Mujer | 3 |
| MEZZANO, SERGIO | Hombre | 1 |
| KRALL, PAOLA | Mujer | 2 |
| Institución | # Pub |
|---|---|
| Centro De Estudios Científicos | 3 |
| Univ Miami | 2 |
| University Of Miami Leonard M. Miller School Of Medicine | 2 |
| Universidad Austral De Chile | 1 |
| Año | Firma | Institución (Incites asoc.) | H Index | Average Percentile | Impact Citation | Impact Relative World | Impact Journal Normalized Citation | Impact Category Normalized Citation | Percentage Cited | Percentage Top 1 | Percentage Top 10 | Percentage Journal Q1 | Percentage Journal Q2 | Percentage Journal Q3 | Percentage Journal Q4 |
|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|
| 2019 | Canales, Cesar P. | Universidad Austral de Chile | 1.0 | 18.6 | 53.0 | 5.9 | 0.8 | 1.3 | 100.0 | 0.0 | 0.0 | 100.0 | 0.0 | 0.0 | 0.0 |
| Título | Año | Citas | Tipo | Revista | Indexada |
|---|---|---|---|---|---|
| Podocyte Specific Overexpression Of Wild Type Or Mutant Trpc6 In Mice Is Sufficient To Cause Glomerular Disease | 2010 | 85 | artículo de investigación | P Lo S One | WoS Scopus |
| Functional And Cellular Characterization Of Human Retinoic Acid Induced 1 (Rai1) Mutations Associated With Smith Magenis Syndrome | 2010 | 22 | artículo de investigación | Bmc Molecular Biology | WoS Scopus |
| Abnormal Social Behaviors And Altered Gene Expression Rates In A Mouse Model For Potocki Lupski Syndrome | 2008 | 65 | artículo de investigación | Human Molecular Genetics | WoS Scopus |
| Palabra Clave | #Pub |
|---|---|
| g-protein | 1 |
| transient receptor | 1 |
| transgenic mice | 1 |
| smith-magenis-syndrome | 1 |
| slit diaphragm | 1 |
| rett-syndrome | 1 |
| rearrangements | 1 |
| rai1 | 1 |
| phenotype | 1 |
| nephrotic syndrome | 1 |
| mutations | 1 |
| mouse models | 1 |
| mice | 1 |
| homologous recombination | 1 |
| gene | 1 |
| 17p11.2 | 1 |
| focal segmental glomerulosclerosis | 1 |
| expression | 1 |
| duplication | 1 |
| dup(17)(p11.2p11.2) | 1 |
| critical interval | 1 |
| cre recombinase | 1 |
| copy number | 1 |
| circadian-rhythm | 1 |
| chromosomal rearrangements | 1 |
| cation channel | 1 |
| behavior | 1 |
| autism | 1 |
| animal-models | 1 |
| 17p11.2 deletions | 1 |